Xanthomonas campestris

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		+	{| border="1" style="text-align: left;"
		+	|+ '''Scientific classification'''
		+	!Kingdom || Eubacteria
		+	|-
		+	! Phylum || Proteobacteria
		+	|-
		+	! Class || Gamma proteobacteria
		+	|-
		+	! Order || Xanthomonadales
		+	|-
		+	! Family || Xanthomonadaceae
		+	|-
		+	! Genus || Xanthomonas
		+	|-
		+	! Species || '''''X. campestris '''''
		+	|-
		+	! Binomial || ''Xanthomonas campestris''
		+	|}
		+
		+
		+
		+	==Virulence==
		+	Their virulence appears to be due primarily to secreted and surface compounds that could increase host nutrient loss, or avoid or suppress unfavorable conditions in the host. Type II and III secretory pathways are essential for virulence. Some individual extracellular enzymes (type II-secretion dependent) affect final bacterial population levels, whereas some avirulence gene products (type III-secretion dependent) affect virulence by altering host metabolism. Avr proteins, probably secreted via a pilus, can also be recognized by host resistance gene products. Virulence is also associated with bacterial surface polysaccharides, which may help to avoid host defense responses, and regulatory gene systems, which can control virulence gene expression.
		+
		+
		+
		+	==References==
		+	[http://en.wikipedia.org/wiki/Xanthomonas_campestris Wikipedia]

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Revision as of 05:56, 7 July 2010

Xanthomonas campestris

Scientific classification

Kingdom	Eubacteria
Phylum	Proteobacteria
Class	Gamma proteobacteria
Order	Xanthomonadales
Family	Xanthomonadaceae
Genus	Xanthomonas
Species	X. campestris
Binomial	Xanthomonas campestris

Virulence

Their virulence appears to be due primarily to secreted and surface compounds that could increase host nutrient loss, or avoid or suppress unfavorable conditions in the host. Type II and III secretory pathways are essential for virulence. Some individual extracellular enzymes (type II-secretion dependent) affect final bacterial population levels, whereas some avirulence gene products (type III-secretion dependent) affect virulence by altering host metabolism. Avr proteins, probably secreted via a pilus, can also be recognized by host resistance gene products. Virulence is also associated with bacterial surface polysaccharides, which may help to avoid host defense responses, and regulatory gene systems, which can control virulence gene expression.

References

Wikipedia